Immune cell
[대역어] None
[용어속성] Cell
[용어속성] Cell
Comparative miRNA transcriptomics of macaques and mice reveals MYOC is an inhibitor for Cryptococcus neoformans invasion into the brain
원숭이와 생쥐의 비교 miRNA 전사체는 MYOC가 뇌로의 크립토코커스 네오포르만스 침입에 대한 억제제임을 보여줍니다
Article
[키워드] AIDS
Brain
brain dissemination
brains
C. neoforman
C. neoformans
caused
Central nervous system
centric
clinical samples
clinically
CNS
Comparative
Cryptococcal meningoencephalitis
cryptococcosis
Cryptococcus neoforman
Cryptococcus neoformans
cytoskeleton
dissemination
enhancer
facilitate
fungal
Fungal infection
help
HIV/AIDS
HIV/AIDS patient
HIV/AIDS patients
host-pathogen interactions
hosts
human pathogen
Immune cell
immune cells
individuals
Infection
inhibitor
inhibitors
Invasion
macaque
Macrophage
mechanism
meningoencephalitis
mice
miRNA
miRNA transcriptome.
MYOC
myocilin
Pathogenic microorganisms
pathway
Patient
patients with COVID-19
performed
Prevalence
regulated
response
reveal
Transcriptome
transcriptomics
Trojan horse
[DOI] 10.1080/22221751.2022.2081619 PMC 바로가기 [Article Type] Article
[DOI] 10.1080/22221751.2022.2081619 PMC 바로가기 [Article Type] Article
Induction of pulmonary HLA-G expression by SARS-CoV-2 infection
Article
[키워드] accompanied
Antigen
Autopsy
biopsies
CD8
Cell
changes in
correlated
distribution
epithelial cell
Evidence
expressed
expression
expression level
Frequency
heart muscle
HLA-G
Hyperactivation
Immune cell
Immune cell infiltration
immune cells
Immune escape
immune response
induction
information
kidney
leukocyte
lung
lung alveolar
lung epithelia
lung epithelial cell
lung tissue
Macrophage
marker
microRNA
microRNA.
miRNA
miRNA expression
modulating
organs
other organ
patients
protein expression
SARS-CoV-2
SARS-COV-2 infection
SARS-CoV-2 infections
SARS-CoV-2 nucleocapsid
SARS-CoV-2-
SARS-CoV-2-infected patient
specimen
Spike protein
surgical
T cell responses
T cells
therapeutic target
tissue
tissues
Treatment
Tumor
virus-infected cells
while
[DOI] 10.1007/s00018-022-04592-9 PMC 바로가기
[DOI] 10.1007/s00018-022-04592-9 PMC 바로가기
Immunoassay and mass cytometry revealed immunological profiles induced by inactivated BBIBP COVID-19 vaccine
Article
[키워드] analyzed
antibody
Antibody titer
B cell
booster
booster dose
caused
Cell
Cluster
Concentration
conducted
COVID-19
COVID-19 vaccine
CyTOF
Cytometry
declined
decrease
distribution
Efficacy
ENhance
healthy
highlight
humoral
Immune cell
immune response
Immunity
immunization
immunoassay
immunoassay.
immunological profile
inactivated
individual
injection
Injections
lack
maintain
marker
multicenter
neutralizing antibody
not significantly different
participant
prevalence of COVID-19
proportion
protective effect
recall
receiving
recruited
reduced
SARS-CoV-2
second dose
serum antibody
shown
significantly
significantly increased
Still
subject
surface marker
sVNT
the antibody response
the vaccine
vaccination
Vaccine
vaccine doses
vaccine injection
Viral variants
virus neutralization test
[DOI] 10.1002/jmv.27983 PMC 바로가기
[DOI] 10.1002/jmv.27983 PMC 바로가기
Single-cell transcriptomic atlas reveals distinct immunological responses between COVID-19 vaccine and natural SARS-CoV-2 infection
Article
[키워드] Activation
acute respiratory syndrome
alteration
analysis
antibody production
Apoptosis
B cell
caused
collected
CoronaVac
coronavirus
COVID-19
COVID-19 infected patient
COVID-19 patients
COVID-19 vaccine
cytotoxicity
downregulation
Emergency use authorization
expression
expression of gene
feature
greater
HLA class II
IL21R
immune
Immune cell
immune cell types
immune response
immunization
immunized
immunological
Immunological response
immunological responses
inactivated
Inactivated vaccine
individual
individuals
Infection
Inflammatory cytokine
innate immune cell
interferon response
magnitude
mechanism
Migration
mononuclear cells
naïve B cell
naïve B cells
pandemic
PBMC
Peripheral blood
phenotype
plasma
profile
Proinflammatory cytokine
proportion
provide
reveal
SARS-CoV-2
SARS-COV-2 infection
scRNA-seq
Sequencing
severe disease
single-cell
Single-cell RNA sequencing
single-cell sequencing.
Sinovac
systemic immune response
T cell
thrombosis
transcriptomic
upregulation
vaccine-induced immune response
[DOI] 10.1002/jmv.28012 PMC 바로가기
[DOI] 10.1002/jmv.28012 PMC 바로가기
Evolving understanding of antibody-dependent enhancement (ADE) of SARS-CoV-2
Article
[키워드] ADE
antibody
Antibody-dependent enhancement
antibody-dependent enhancement (ADE)
complex
countermeasures
explain
feeble
immune
Immune cell
immune system
in vivo
internal environment
Interpretation
mechanism
multiple roles of macrophages
Mutant strains
neutralize
patients
Perspective
receptor-mediated.
reduced
SARS-CoV-2
SARS-COV-2 infection
shown
specific antibody
virus
[DOI] 10.3389/fimmu.2022.1008285 PMC 바로가기
[DOI] 10.3389/fimmu.2022.1008285 PMC 바로가기
Reconstructed Genome-Scale Metabolic Model Characterizes Adaptive Metabolic Flux Changes in Peripheral Blood Mononuclear Cells in Severe COVID-19 Patients
Article
Published on
Journal: International Journal of Molecular Sciences
[Category] COVID19(2023년), SARS, 유전자 메커니즘, 치료제,
Journal: International Journal of Molecular Sciences
[Category] COVID19(2023년), SARS, 유전자 메커니즘, 치료제,
[키워드] acute respiratory syndrome
adaptive
aerobic
Analysis
anti-inflammatory effect
antiviral drugs
biogenesis
caused
change
changes in
Coronavirus disease 2019
Coronavirus-2
COVID-19
differentially expressed
enzyme
FASN
fatty
flux
Glycolysis
Health
Immune cell
Inflammation
Inflammatory
lipids
mechanism
metabolic
metabolic model
metabolism
metabolism-directed therapy.
metabolites
Model
mononuclear cell
nucleotide
oxidative phosphorylation
pathogenic
PBMC
PBMCs
peripheral
Peripheral blood
potential therapeutic targets
provide
reaction
reconstructed
Regulation
responsible
retinol
SARS-CoV-2
severe COVID-19 patients
SLC2A3
therapy
Transcriptome
Treatment
Vitamin
[DOI] 10.3390/ijms232012400 PMC 바로가기
[DOI] 10.3390/ijms232012400 PMC 바로가기
Social connection and gene regulation during the COVID-19 pandemic: Divergent patterns for online and in-person interaction
Article
[키워드] absence
analyzed
antibody production
antiviral immunity
antiviral response
association
blood sample
characterized
connection
conserved
COVID-19
COVID-19 pandemic
CTRA
disease risk
down-regulation
Effect
eudaimonia
examined
experience
Factors
female
Gene Expression
Health
healthy
Immune cell
immune system
Interaction
Linear model
loneliness
measure
Multiple
offset
Online social interaction
profile
proinflammatory gene
quantified
question
reduced
Regulation
RNA
Social genomics.
social interaction
transcriptional response
[DOI] 10.1016/j.psyneuen.2022.105885 PMC 바로가기
[DOI] 10.1016/j.psyneuen.2022.105885 PMC 바로가기
SARS-CoV-2 infection produces chronic pulmonary epithelial and immune cell dysfunction with fibrosis in mice
Article
[키워드] acute respiratory distress
acute respiratory syndrome
alveolar
Antiviral
cells
chronic disease
chronic inflammation
clinical recovery
collagen
composed
coronavirus
coronavirus disease
COVID-19
develop
disease severity
dysfunction
dysregulated
EIDD-2801
epithelial
fibroblasts
fibrosis
histologic finding
humans
identify
Immune cell
individual
Intervention
lack
lesion
longitudinal
lung abnormality
lymphoid
mice
mouse-adapted SARS-CoV-2
murine model
Nintedanib
PASC
Pathogenesis
pathway
Population
proliferative
provide
pulmonary disease
reduced
regions
SARS-CoV-2
SARS-COV-2 infection
structures
subpleural
subset
syndrome
tissue samples
transcriptional
virus clearance
[DOI] 10.1126/scitranslmed.abo5070 PMC 바로가기
[DOI] 10.1126/scitranslmed.abo5070 PMC 바로가기
Targeting Doublecortin-Like Kinase 1 (DCLK1)-Regulated SARS-CoV-2 Pathogenesis in COVID-19
Article
[키워드] Activation
amplified
amplify
analyzed
blocked
Blood
Cell
chemokine
chronic liver disease
Chronic Liver Diseases
circulating
comorbid disease
Comorbidity
contribute
controls
correlated
COVID-19
COVID-19 patients
Critical
cytokine
cytokine secretion
DCLK1
DDCLK1
decrease
disease severity
dysregulation
expression
Factor
Factors
Gain
growth factor
hospitalized patient
Host
Hyperinflammatory response
immune
Immune cell
immune response
immunosuppressive
in viral
in vitro
infected cells
Inflammation
Inflammatory cytokine
inflammatory pathway
inhibition
inhibitor
Innate immunity
involved
kinase
Live virus
liver
livers
loss of function
lung
Lung disease
Macrophage
mononuclear cell
mononuclear cells
Mortality
neutrophil
normalized
Pathogenesis
pathology
Patient
Peripheral blood
potential therapeutic target
Prevalence
pro-inflammatory
profile
proinflammatory
promoted
promoter
Protein
S100A9
SARS-CoV-2
SARS-CoV-2-infected cell
secretion
severity of COVID-19
Signaling
subject
targeting
Treatment
Viremia
virus production
virus replication
Virus replication.
with COVID-19
β-catenin
[DOI] 10.1128/jvi.00967-22 PMC 바로가기
[DOI] 10.1128/jvi.00967-22 PMC 바로가기